There are well over 100 options that offer the opportunity to reverse symptoms of Parkinson’s disease using natural methods and therapies. Studies have revently proposed to treat Parkinsons wtih Cinnamon of all things. In fact, there are so many good ideas out there these days that making a choice of which ones to pursue can be daunting to say the least.

Of All Herbs – Cinnamon?

I have an idea for you to consider (among the many possibilities) that attracted my interest. Researchers found success using ground up Ceylon cinnamon in rat studies. I know. I know. It does sound a little silly that a spice like cinnamon might make a difference and humans as research subjects are a far cry from mice. I also know that it will take many years for research studies with humans to be run, if ever.

Why should cinnamon of all spices help reverse Parkinson’s symptoms in mice? Researchers at Rush University report that “It is known that some important proteins like Parkin and DJ-1 decrease in the brain of PD patients.” The Rush study researchers report that ground cinnamon metabolizes into a substance known as sodium benzoate, which helps stop the loss of Parkin and DJ-1 proteins, protect neurons, normalize neurotransmitter levels, and improve motor functions in mice with Parkinson’s symptoms. That is a pretty impressive list, even for mice, Eh?

Why not step aside from the details of the academic research and simply ask yourself – do you like cinnamon? Have you always liked cinnamon? If so, why not make it a point to add a little extra Ceylon cinnamon to spice up your food? Who knows. Maybe your body will thank you profusely.

Research on Cinnamon and Parkinsons

J Biochem Mol Toxicol. 2021 Apr;35(4):e22720. Neuroprotective potential of cinnamon and its metabolites in Parkinson’s disease: Mechanistic insights, limitations, and novel therapeutic opportunities

Abstract

Parkinson’s disease (PD) is the most common neurodegenerative movement disorder with obscure etiology and no disease-modifying therapy to date. Hence, novel, safe, and low cost-effective approaches employing medicinal plants are currently receiving increased attention.

A growing body of evidence has revealed that cinnamon, being widely used as a spice of unique flavor and aroma, may exert neuroprotective effects in several neurodegenerative diseases, including PD.

In vitro evidence has indicated that the essential oils of Cinnamomum species, mainly cinnamaldehyde and sodium benzoate may protect against oxidative stress-induced cell death, reactive oxygen species generation, and autophagy dysregulation, thus acting in a potentially neuroprotective manner.

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Antioxidants (Basel). 2026 Feb 2;15(2):195. Preventive Effects of Cinnamon Leaf Nanosuspension and Byproducts on Type II Diabetes and Parkinson’s Disease in Rat Models

Abstract

Cinnamon leaves, an important source of cinnamaldehyde, have been shown to possess various pharmacological functions. A liposome-derived nanosuspension (CN), hydrosol (CH) and powder in water (CP) prepared from cinnamon leaves were explored for their preventive effects on type II diabetes (T2D) and Parkinson’s disease (PD) in rat models.

Fifteen compounds were determined by UPLC-MS/MS, with cinnamaldehyde being predominant. CN with a mean particle size at 22.6 nm was prepared by mixing extract, lecithin, Tween 80, soybean oil and water with an optimal ratio, while hydrosol was prepared by steam distillation.

A high storage and gastrointestinal stability were observed for CN. Rats were pre-fed with CN, CH and CP separately for 4 weeks, followed by induction with T2D or PD, with all three groups showing better preventive effects on T2D given the number of injections for T2D induction. Additionally, the OGTT, HOMA-IR, TXB2 and aPTT levels were significantly higher in the induction group than in the CN, CH and CP groups, revealing an effective prevention of T2D and cardiovascular disease.

For PD prevention, CN was the most effective in improving muscle stiffness based on the catalepsy test and elevation of dopamine, tyrosine hydroxylase, serotonin, mitochondrial DNA and antioxidant enzymes, accompanied by a decline in alpha synuclein and malondialdehyde.

Robert Rodgers PhD
Parkinsons Recovery®
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